Right Kidney Non functioning
Discussion in 'All Categories' started by Irshad - Feb 19th, 2013 1:25 am.
Irshad
Irshad
My father's blood serum creatinine level was detected as 4.0 md/dl and blood urea level as 132mg/dl two days back. there was no history of any infection or cold. but he was dehydrated and had few vomitings prior to the lab tests.

his blood pressure is 160/90 and is on the medication of stamlo (amlodipine) 5mg once daily. and losar h ( losartan + hydrochlorthiazide ) 5gm once daily.

he had recurrent stroke with right hemiparesis, renal calculi with hydronephrosis ( right kidney) 6 months back and was treated accordingly.,

on ultra sound, the right kidney is having 12-14mm multiple calculi and is declared non functioning.
left kidney is 80% functioning.

his recent condition:
he is not having any vomiting now,
his appetite is low,
fluid input is 1.5 - 2 lit
urine out put is around 1- 1.5lit
urine is clear,
No abdomen pain,
no fever,

he is on antiplatelets and anti coagulants, iron supplements, resourse dialysis powder.
re: Right Kidney Non functioning by Dr M K Gupta - Feb 22nd, 2013 11:31 am
#1
Dr M K Gupta
Dr M K Gupta
Dear Irshad

Chronic renal insufficiency ultimately culminating in end-stage renal disease requiring dialysis or transplantation is a major health issue. The 1st task confronting the doctor caring for a patient with renal disease would be to decide whether or not the renal insufficiency is acute or chronic. The original differential diagnostic procedure for chronic renal insufficiency is made up of determining whether the patient has glomerular disease or interstitial or vascular disease on such basis as a careful history taking, urinalysis, and measurement of 24-hour protein excretion.

Further refinement of diagnostic considerations often requires serologic studies, renal biopsy, or imaging the urinary system with ultrasonography or computed tomography. Management considerations start with the identification and correction from a acute reversible factors behind renal insufficiency in patients with chronic renal disease. Recent reports have shown that effective antihypertensive therapy, especially with angiotensin-converting enzyme inhibitors, restriction of dietary protein, and ideal glycemic control in patients with diabetes, can retard the growth of chronic renal disease. Once these therapeutic strategies are in place, you should anticipate and treat the multiple manifestations of chronic progressive renal insufficiency: fluid overload, hyperkalemia, metabolic acidosis, abnormalities of calcium, phosphorus, and vitamin D metabolism, and anemia.

With regard

M K Gupta

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